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The thyroid dyshormonogenesis in nrf2a Δ5 homozygous mutant embryos is a cell-autonomous phenotype Although the initial characterization of the nrf2aΔ5 mutant phenotype indicates a role of nrf2a during thyroid functional maturation, we could not rule out the possibility that this phenotype is secondary to another defect affecting the whole zebrafish embryos

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Suggestion Further research using similar study protocols is necessary to investigate the hemolysis marker and oxidative stress for those having transfusion every 2 weeks and pretransfusion Hb between 9 to 10.5 g/dL for longer study period