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Aβ plaques result from the abnormal cleavage of amyloid precursor protein (APP) by beta‐secretase (BACE1) and gamma‐secretase, leading to the production of toxic Aβ peptides, particularly Aβ42, which aggregates and forms insoluble plaques

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Molecular characterization of the humoral responses to Cryptococcus neoformans infection and glucuronoxylomannan-tetanus toxoid conjugate immunization