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For some proteins, apo structures of active, uninhibited enzymes may not be available for autoproteolytic/cannibalistic reasons (e.g., cysteine cathepsins) due to self-proteolysis or autodigestion [127,128]

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Studies in chicken embryos suggest that at least part of the mechanism by which BPC-157 promotes vascular growth is through the stimulation of VEGFR2, a cell surface receptor active in the nitric oxide signaling pathway[4], [7], [8]