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AMPK hyperactivation promotes dendrite retraction, synaptic loss, and neuronal dysfunction in glaucoma

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(iv) given that nmTORC2 regulates histone acetylation (e.g., via acetyl-CoA supply or SIRT6 regulation) [93], combination therapies targeting both mTORC2 and specific epigenetic enzymes (such as histone acetyltransferases or deacetylases) may achieve nuclear-selective effects