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The two most commonly raised markers in vitamin B12 deficiency are homocysteine and methyl malonic acid (MMA)

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This induction is primarily mediated through the activation of the heme-regulated eIF2α kinase (HRI), which leads to increased ATF4 translation and the subsequent activation of genes involved in the stress response, highlighting UTX’s role in modulating cellular stress pathways and its potential impact on cancer progression (Kitajima et al., 2021)