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Inhibition of excessive mitochondrial fission reduced aberrant autophagy and neuronal damage caused by LRRK2 G2019S mutation

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Upon recruitment to developing plaques, monocytes encounter many inflammatory stimuli such as damage associated molecule patterns (DAMP), oxLDL, and IFNγ that trigger inflammatory cascades after engagement with toll-like receptors (TLR), scavenger receptors and cytokine receptors, respectively