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However, it is interesting that glioblastomas expressing PTPRZ1-MET often also express METΔEx14 splice variant, suggesting that MET activation in these tumors may be complex

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The mechanism is mainly through stimulation of the nitric oxide system, with secondary effects on VEGFR-2 (which drives angiogenesis and explains the wound healing), the serotonin and dopamine systems, COX-2, and HO-1