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These cardiac repair studies established TB-500 as one of the most extensively characterised peptide research tools for studying cardiac regeneration biology and provided the foundational evidence for thymosin beta-4’s role as an endogenous cardiac repair mediator and establishing that actin-sequestering peptides have biological significance beyond cytoskeletal regulation in the context of cardiac injury response
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ROS-induced impairment of the IFN response occurs in murine herpes virus infection, with the inhibition taking place downstream of cytoplasmic DNA sensing 59
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